Fwd: EE PhD Oral Examination - Rostam Dinyari, Thursday, October 21, 2010; 3:45 pm

Rostam Dinyari rostam at stanford.edu
Tue Oct 19 13:57:34 PDT 2010

---------- Forwarded message ----------
From: Natasha Newson <nnewson at stanford.edu>
Date: Tue, Oct 19, 2010 at 1:37 PM
Subject: EE PhD Oral Examination - Rostam Dinyari, Thursday, October 21,
2010; 3:45 pm
To: ee-students at mailman.stanford.edu



Student:  Rostam Dinyari
Adviser:   Peter Peumans

Date:       Thu, Oct 21, 2010
Time:       3:45 PM (Refreshments served at 3:30 PM)
Location: Allen-X 101 Auditorium


In age-related macular degeneration (AMD) and retinitis pigmentosa (RP), two
leading causes of blindness, the photoreceptor layer of the retina is
degenerated while the other layers remain functional. The function of
photoreceptors is very similar to that of solar cells. Upon receiving the
light, they stimulate the inner layers of retina electrically and
chemically. These data are then processed and compressed by a complex
circuit of retinal neurons - horizontal cells, biopolar cells, amacrine
cells, and ganglion cells - and sent to the brain for recognition.

We have developed a monolithic silicon photovoltaic retinal implant that can
replace the degenerated photoreceptor layer. The implant requires no
electrical power or data connection. It consists of a two-dimensional
network of miniature silicon solar cells that directly stimulate the retina
when illuminated by a goggle-IR laser system. A MEMS process isolates
adjacent pixels and makes the arrays curvable allowing them to conform to
the shape of the retina and allow for transfer of nutrients to the remaining
layers of retina. The MEMS technique also provides isolation between the
bodies of the three series-connected subpixels that make up each pixel. A
high spatial resolution, a large field of view, and absence of mechanical
strain are all achieved as a result of this approach.

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